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7. Mich'l O'Rorque
8. James O'Grady

2 years,

Chicago..
Chicago.
Chicago.

Dr. E. W. Lee. Nov. 23, 1877.
Dr. E. W. Lee. Dec. 7, 1877.
Dr. E. W. Lee. March 18, 1878.

2 mos.
2 years,
4 mos.

croup.

croup.
Membranous Recovered.

Membranous Recovered. 94 Brown St., Dr. E. W. Lee. Oct. 12, 1878. croup, no

membrane

visible.

Chicago.

9. Albert Caproni.. 4 years.

Diphtheria.

Died.

254 S. Halsted Dr. E. W. Lee. St., Chicago.

Dec. 9, 1878.

REMARKS.

Sick three days. Used chloroform.
Lived thirty hours.

Sick two days. Asphyxia immi-
nent. Gave chloroform. Lived two
and a half days. Died by asphyxia.
Two weeks complaining. Chloro-
form. Artificial respiration necessary
after the operation. Removed tube
sixth day.

Sick thirty-six hours. Lived two
and a half days. Died by exhaus-
tion. Chloroform.

Sick five days. Chloroform. Lived
twelve hours; died of exhaustion.
Sick four days. Lived four days.
Died from asphyxia. Chloroform.
Sick two days. Removed tube
fourth day. Chloroform.

Three days sick. Tube worn nine
days. Chloroform. Condition:
Pulse, 140; respiration, 48; tempera-
ture, 103.5° F.; laryngeal obstruc-
tion permanent forty-eight hours;
whispering voice; nasal dilatation;
retrocession of the base of the thorax
marked; slight glandular enlarge-
ment. Low operation. Anesthetic
difficult of toleration.

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12. Aaron West..

5 years.

Diphtheria. Recovered. S. Dearborn Dr. E. W. Lee. Aug. 17, 1879.
St., Chicago.

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husky voice; dilated nostrils; epi-
gastric sinking; glandular enlarge-
ment in neck. Low operation.
Four days sick. Tube worn and
lived twenty-six hours. Cause of
death, exhaustion. Chloroform.
Condition: "Asphyxiated, death im-
minent.' Low operation. Parents
grateful that the operation was done.
Should have been done earlier. Diph-
theria suspected.

Lived sixty hours after operation.
Immediate cause of death, asphyxia
caused by extension of membrane.
Anesthetic, chloroform. Impending
suffocation at time of operation.
Low operation.

Duration of previous illness, five
days. Tube worn one week. Pulse,
160; respiration, 60; temperature,
103° F. Low operation.

Duration of previous illness, one
week. Lived fourteen hours after
operation.
Immediate cause of

death, exhaustion. Anesthetic,
chloroform. Pulse, 168; tempera-
ture, 103.5° F. Low operation.
Duration of previous illness, three
days. Lived twenty-four hours after
the operation. Immediate cause of
death, asphyxia. Anesthetic, none.
Pulseless, deeply cyanosed. Respira-
tion almost ceased. Low operation
rapidly performed.

Previous illness, eight days. Lived
six days. Cause of death, pneumonia
Anesthetic, chloroform. Pulse, 144;

respiration, 48; temperature, 103° F. Low operation.

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REMARKS.

Previous illness, seven days. Tube
worn seven days. Anesthetic, chloro-
form. Pulse, 144; respiration, 64;
temperature, 101° F.
Exudation of
membrane over the soft parts of
palate and tonsils. Low operation.

Previously ill, ten days. Tube worn
five days. Anesthetic, chloroform.
At time of the operation: Pulse, 132;
respiration, 48; temperature, 102° F.
Head thrown back, lips blue, face
pale; epigastric retrogression. Two
hours after operation: Pulse, 112;
temperature, 101° F.; respiration, 36.
Breathing with perfect freedom. High
operation.

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Previous illness, eight days. Lived
four hours after the operation.
mediate cause of death, collapse,
hemorrhage. Severe dyspnea, ex-

tensive diphtheric exudation. Marked symptoms of blood-poisoning. Anesthetic, chloroform.

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tions prominently two varieties of tracheal constriction. The first variety, although rare, has, however, been demonstrated in a certain number of cases. It arises through the presence of fleshy growths springing from the wound, especially through those deeply seated upon the borders of the tracheal incision, and which grow in the midst of a cicatricial tissue projecting into the air-passages after the closure of the cutaneous wound. The second variety, up to the present time, has not been described at all.

A tracheotomized child was seized with a fit of suffocation just as the physician was attempting to effect a permanent removal of the cannula. Examining the depths of the tracheal wound, he perceived a reddish prominence in the interior of the trachea, which was taken for fleshy vegetation of the posterior wall. The child died in a fit of suffocation. Professor Guyon recognized, upon the postmortem specimen sent him, that the projection regarded during life as vegetation was formed by the posterior wall of the trachea itself, which was folded longitudinally in its entire thickness. This folding was itself due to the approximation of the posterior extremities of the tracheal rings, separated anteriorly for the introduction of the cannula.

M. Currie, experimenting with the view of discovering the conditions of the production of this protrusion, concluded that this particular variety of constriction, which hitherto had not been pointed out, ought to be, nevertheless, rather frequent among children. It occurs after the introduction of the cannula, and the more readily according as the membranous span which lies between the posterior extremities of the rings is large. It affects chiefly first the three rings of the trachea. The projection which results produces a tracheal constriction that may persist and prove a permanent obstacle to the removal of the cannula.

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THE ENDOSCOPE IN THE LOCAL TREATMENT OF CHRONIC GONORRHEA OR GLEET AND GONORRHEAL RHEUMATISM*

WITH A. HINDE

I. THE ENDOSCOPE

It was in the first quarter of the present century that the experiment was first made to render visible the urethral canal from its meatus to the bladder by Barini, in Frankfort, in 1806; Fisher, in Boston, in 1824; Segalas, in Paris, in 1826; and Hacken, in Riga. These experimenters failed to attract professional attention.

To Desormeaux, after years of arduous toil in this field of investigation, we owe the credit of successful accomplishment. In 1852 he exhibited his endoscope in the French Academy. In 1865 he published his valuable monograph† on this subject, in which he advanced a series of facts and original observations which have opened up a new path in the diagnosis and treatment of urethral diseases.

Desormeaux's instrument is too well known to need description here, therefore we shall mention only a few details, showing some defects in his original instrument. Desormeaux's lamp gave insufficient light, and on this account Bruns used the calcium light, and Dr. Andrews, of Chicago, the magnesium light. These modifications were too complicated and hence were not generally used. At the present time kerosene or kerosene and camphor forms the burning material for the lamp. Desormeaux's lamp is connected with a tube containing a reflector and telescope; to the end of the latter are connected the urethral tubes. Desormeaux's instrument is too costly-one hundred and fifty francs (twentyfive to thirty dollars) and also too complicated for general use, and consequently simplified instruments have been invented by Warwick‡ and Wales, but their instruments have not come into general use, and Fürstenheim pronounces them inefficient.

After several years of experimentation with a view of simplifying the endoscope I decided upon the use of the following instrument:

1. For the lamp, a circular wick; ordinary kerosene is all sufficient. 2. A common laryngoscopic reflector is also needed, but if sunlight can be obtained, a plain reflector will best answer our purpose.

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† De l'endoscope et de ses applications au diagnostic et au traitement des affections de l'urethre et de la vessie, Paris, 1865.

Brit. Med. Jour., 1867, vol. ii, p. 124.

§ Virchow's Jahresbericht, 1868, vol. ii, p. 180.

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